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level: CLASSIFICATION OF DISEASE

Questions and Answers List

level questions: CLASSIFICATION OF DISEASE

QuestionAnswer
A clinical diagnostic category in which the pulp is symptom free and normally responsive to pulp testingNormal pulp
A clinical diagnosis based on subjective and objective findings indicating that the inflammation should resolve and the pulp return to normal.Reversible Pulpitis
A clinical diagnosis: lingering thermal pain, spontaneous pain, referred pain.Symptomatic Irreversible Pulpitis
A clinical diagnosis based on subjective and objective findings indicating that the vital inflamed pulp is incapable of healing.Asymptomatic Irreversible Pulpitis
A clinical diagnosis: no clinical symptoms but inflammation produced by caries, caries excavation, trauma.Asymptomatic Irreversible Pulpitis
A clinical diagnostic category indicating death of the dental pulp. The pulp is usually nonresponsive to pulp testing.Pulp Necrosis
A mild to moderate inflammation of the pulp caused by noxious stimuli in which the pulp is capable of returning to the uninflamed state following the removal of stimuliReversible Pulpitis
“reactive” form of pulpitis; wherein the dentin has the ability to form reparative dentin to protect the pulp from noxious stimuliReversible Pulpitis
Symptoms: Thermal - momentary, quick, sharp, hypersensitive response that subsides as soon as stimulus is removed; Percussion (-); Palpation (-)Reversible Pulpitis
Types of Reversible Pulpitis:Characterized by a sharp pain lasting for a short moment onlyAcute Reversible Pulpitis
Types of Reversible Pulpitis:Does not occur spontaneously, and must have stimulus to produce pain; pain ceases upon removal of the stimulus; cause of pain is generally traceable to a stimulusAcute Reversible Pulpitis
Types of Reversible Pulpitis:May result from incipient caries and is resolved upon removal of the caries and proper restorationChronic Reversible Pulpitis
most common cause of asymptomatic reversible pulpitisBacteria from caries
Persistent inflammation of the pulp which is caused by noxious stimuliIrreversible Pulpitis
Is usually caused by hot or cold stimuli; Can occur spontaneously; Is persistent for several minutes to hours even after removal of thermal stimulusIrreversible Pulpitis
Types of Irreversible Pulpitis:Pulp exhibits little or no pain, except when food is packed into the cavityChronic Irreversible Pulpitis
Types of Irreversible Pulpitis:Symptoms include a diffuse, dull, constant pain, characterized as throbbing and gnawingChronic Irreversible Pulpitis
Types of Irreversible Pulpitis:extreme pain when subjected to heatChronic Irreversible Pulpitis
Types of Irreversible Pulpitis:Episodes of pain due to sudden temperature change; Sudden temperature changes, particularly coldAcute Irreversible Pulpitis
Types of Irreversible Pulpitis:Recumbency (lying down) which results in congestion of the blood vessels of the pulpAcute Irreversible Pulpitis
Types of Irreversible Pulpitis:Sweet or acidic food, Pressure from packing food into a cavity, Suction exerted by the tongue or cheekAcute Irreversible Pulpitis
Types of Irreversible Pulpitis:Localized referred pain which lingers after the stimulus is removedAcute Irreversible Pulpitis
refers to the death of the pulpPulp Necrosis
described as a dry-gangrenous appearancePulp Necrosis
Thermal (-), EPT (-), Percussion (-) or (+),Pulp Necrosis
Two types of Pulp Necrosis:Happens when the soluble portion of the pulp is precipitated or is converted into a solid materialCoagulation Necrosis
Two types of Pulp Necrosis:Yellowish in colorCoagulation Necrosis
Two types of Pulp Necrosis:Most common pulp necrosis observedLiquefaction Necrosis
Two types of Pulp Necrosis:Bavarian appearanceLiquefaction Necrosis
Two types of Pulp Necrosis:results when pulp tissue is converted into a softened mass, a liquid, or an amorphous debris by the proteolytic enzymes released by caries bacteriaLiquefaction Necrosis
Two types of Pulp Necrosis:grayish or brownish discoloration and the tooth lacks the usual brilliance and lusterLiquefaction Necrosis
Clinical Classification of Periapical Diseases:Inflammation around the apex of vital or non-vital teeth; Slightly to extremely painful when chewingACUTE APICAL PERIODONTITIS
Clinical Classification of Periapical Diseases:Usually observed upon percussion; Radiograph shows slightly widened periodontal ligamenACUTE APICAL PERIODONTITIS
Clinical Classification of Periapical Diseases:Cause: premature contacts; periodontal in originACUTE APICAL PERIODONTITIS
Clinical Classification of Periapical Diseases: Inflammation and destruction of apical periodontium that is of pulpal originCHRONIC APICAL PERIODONTITIS
Clinical Classification of Periapical Diseases:Appears as an apical radiolucent area, and does not produce clinical symptoms; no painCHRONIC APICAL PERIODONTITIS
Clinical Classification of Periapical Diseases:Tooth feels “different or hollow” when chewing or with percussionCHRONIC APICAL PERIODONTITIS
Clinical Classification of Periapical Diseases:75% is associated to granuloma; Clinically shows swelling on the gingiva, but not painful and also no sinus tractCHRONIC APICAL PERIODONTITIS
Clinical Classification of Periapical Diseases:Thermal and EPT – negative; Percussion – slight; Palpation – slightApical Periodontitis
Clinical Classification of Periapical Diseases:Swelling, Bukol sa gums, Mild - moderateAcute Periradicular Abscess
Clinical Classification of Periapical Diseases:Mobile and extruded, Painful when bitingAcute Periradicular Abscess
Clinical Classification of Periapical Diseases:With/ without Sinus tract, No pain, Necrotic toothChronic Periradicular Abscess
Clinical Classification of Periapical Diseases:Gumboil, with or without bukolChronic Periradicular Abscess
Clinical Classification of Periapical Diseases:Acute exacerbation of a chronic lesionPhoenix Abscess
Clinical Classification of Periapical Diseases:Acute before but with increased immunity, pain disappeared but with an event where immunity is decreased, pain reappearsPhoenix Abscess
Clinical Classification of Periapical Diseases:Comes and goes, and there is no presence of sinus tract, making this case difficult to treat; Reaction of bone to traumaPeriapical osteosclerosis