For the 3 compensatory mechanisms for hypovolemic shock, what is the pathophysiology behind that

Increased sympathetic activity --> Increase HR, Increase contractility, Vasoconstriction , Venoconstriction RAAS --> angiotensin II leads to aldosterone release, Na+ reabsorption , vasoconstriction , releases noradrenaline to increase sympathetic activity and release of ADH from the anterior pituitary gland Internal Transfusion --> Increase in TPR leads to decrease hydrostatic pressure of the capillary which leads to net flow of fluid into the capillaries