How are cardiac thrombosis?

MI can predispose mural cardiac thrombus by dyskinetic myocardial contraction and endocardial damage, rheumatic heart disease may cause arterial wall thrombus causing atrial dilation /fibrillation. Cardiac and aortic walls are prone to embolization, brain, kidneys, spleen are more likely targets of embolization. We could also have intracardiac thrombus, triggered by stasis in left atrium upstream of mitral stenosis (atrial stenosis in atrial fibrillation). Often cause of parietal factor (mural thrombus on area of MI/ heart valves altered by bacterial infection (vegetations), prosthetic valves.) Main risk of progression is embolism. Cardiac valve thrombus (may get sterile vegetations on uninfected valves in states of hypercoagulability (non-bacterial thrombotic endocarditis), less common sterile warty endocarditis (Libman -Sacks endo) in SLE)