ORLPTD1
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🇬🇧 | 🇬🇧 |
Oral pathology discusses the ____ of the tooth | Hard tissues |
Missing tooth (genetic) | Anodontia |
Diabetes is manifested in the oral cavity as | Periodontitis/ gingivitis |
Cells react to conditions that are not normal by adapting to the change | CELLULAR ADAPTATION |
Underdevelopment of an organ | Hypoplasia |
Organ is present but not developed properly/fully thereby it is not functioning to its capacity | Hypoplasia |
Most common cause of atrophy | Ischemia |
Refers to metabolic disturbances and deterioration of the cells or the area immediately surrounding the cells | Degenerative changes |
Cellular self-destruction | Apoptosis |
Increase inflow of the blood to an area | Active hyperemia |
Decreased outflow of blood with stagnation of venous current. | Passive hyperemia |
Localized tissue death | Infarct |
Infarcts are usually caused by | Fat deposits or blood clots |
Causative factors of the disease | Etiology |
Example of singular disease | Tuberculosis |
Example of multifactorial disease | Hypertension |
Conditions within or around the organism or host that do not inhibit the action of pathogenic agents. | Susceptibility |
Condition of the host (intrinsically) that makes them at risk of being affected by certain conditions | Susceptibility |
When an individual is compromised by one disease or condition, there is a much higher risk of developing a second disease or condition. | Pre-existing Condition |
Condition that the patient has already and will worsen his condition upon application of another stimuli | Pre-existing Condition |
Tay Sach's predominantly affects | Jewish descendants |
Sickle-cell Anemia predominantly affects | American-African lineage |
EVENTS IN ACUTE INFLAMMATORY PROCESS: brief hemorrhage control | VASOCONSTRICTION |
EVENTS IN ACUTE INFLAMMATORY PROCESS:Causes gaps in vessel wall between endothelial cells | INCREASED VASCULAR PERMEABILITY |
EVENTS IN ACUTE INFLAMMATORY PROCESS:Begins process of exudate formation and vascular stasis | INCREASED VASCULAR PERMEABILITY |
EVENTS IN ACUTE INFLAMMATORY PROCESS:Leukocytes move to the endothelial walls and begin the process of rolling | VS: MARGINATION |
EVENTS IN ACUTE INFLAMMATORY PROCESS:Leukocytes squeeze through gaps in endothelial cells | VS: TRANSMIGRATION |
EVENTS IN ACUTE INFLAMMATORY PROCESS:Drives PMNs and other leukocytes to the affected area | CHEMOTAXIS |
EVENTS IN ACUTE INFLAMMATORY PROCESS:Enables Ingestion and digestion of foreign material or cellular debris | PHAGOCYTOSIS |
EVENTS IN ACUTE INFLAMMATORY PROCESS:Removes debris through lymphatic system | TERMINATION OF PROCESS |
The body’s mechanism for dealing with the injuries caused by internal and external stimuli. | INFLAMMATORY PROCESS |
Could be beneficial (initiate healing) or detrimental to the body | INFLAMMATORY PROCESS |
Considered to be the cornerstone of pathology | INFLAMMATION |
FACTORS THAT DETERMINE THE DEGREE OF RESPONSE: Type of tissue injury | Severity of the injurious stimuli |
FACTORS THAT DETERMINE THE DEGREE OF RESPONSE: Affects the capability of the host | The reactive capability of the host |
The reactive capability of the host | Resistance & age and physical make up |
TYPES OF INJURY: Produce a burn | THERMAL OR ELECTRICAL |
TYPES OF INJURY: Virus, bacteria, fungal | BIOLOGICAL |
COMPONENTS OF REACTION | Damage to the tissues, Alteration, Exudation, Granulation and Repair |
2 CATEGORIES OF INFLAMMATION: Occasionally can be very extensive and involve multiple body organs or systems | ACUTE INFLAMMATION |
2 CATEGORIES OF INFLAMMATION: most often limited in area and duration and characterized by the Cardinal signs of Inflammation. | ACUTE INFLAMMATION |
5 CATEGORIES OF INFLAMMATION: Inflammation that goes beyond 2 weeks | CHRONIC INFLAMMATION |
Causative agent in inflammatory process of this cardinal sign: dolor | Edema and chemical mediators |
The one that causes edema is _____ or the plasma which exceeds the blood vessels and goes towards the injured area | Serous exudates |
FIBER FORMING CELLS | Fibroblast |
VASOFORMATIVE CELLS | ANGIOBLASTS |
REPAIRS THE BLOOD VESSELS | FIBROBLAST AND ANGIOBLAST |
REPAIR WITH SCARRING | FIBROSIS |